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Program scheduleThursday, July 217:00-8:00 Breakfast
8:00-9:30 Welcome Lecture
SESSION 1: Clinical trials and dilemmas9:30-10:00 Morning Break
10:00-12:00 SESSION 1 Clinical trials and dilemmas (cont.):
12:00-13:00 Lunch
13:00-14:00 KEYNOTE 1: Dr. Tomio Sasaki
14:00-14:30 SESSION 2: Neurocritical care (monitoring, acute Care, imaging)
14:30-15:00 Afternoon Break
15:00-17:00 SESSION 2 (cont): Neurocritical care (monitoring, acute care, imaging)
17:00-18:00 POSTER SESSION
18:00-19:00
Competition, Judging & Reception19:00-21:00 COSBID Oral Session 1
Friday, July 227:00-8:00 Breakfast
8:00-9:30 SESSION 3: Spreading depression and neurovascular coupling
(COSBID Oral Session 2)
9:30-10:00 Morning Break
10:00-12:00 SESSION 3 (cont): Spreading depression and neurovascular coupling
(COSBID Oral Session 3)
12:00-13:00 Lunch13:00-14:00 White Paper Discussion
14:00-14:30 SESSION 4: Inflammation and blood-brain barrier
14:30-15:00 Afternoon Break
15:00-17:00 SESSION 4 (cont) : Inflammation and blood-brain barrier
BID PRESENTATIONS Vasospasm 2013
17:00-18:00 COSBID business meeting
18:00-19:00 DinnerPoster Award Presentation
Saturday, July 237:00-8:00 Breakfast
8:00-9:30 WINNER PRESENTATIONS
SESSION 5: Vascular and microvessel
9:30-10:00 Morning Break
10:00-12:00 SESSION 5 (cont): Vascular and microvessel
12:00-13:00 Lunch
13:00-14:00 KEYNOTE 2: Dr. Constantine Iadecola
SESSION 6: Genetic and environmental factors
14:30-15:00 Afternoon Break
15:00-17:00 SESSION 6 (cont): Genetic and environmental factors
17:00-18:00 Vasospasm 2013 Venue Announcement & Champagne Toast
Program highlightsSAH triggers recurrent waves of sustained depolarization (cortical spreading depression; CSD) that challenge the cortex at all levels from gene transcription to cerebral blood flow. Recent studies have shown that CSD can induce microvascular constriction resulting in oxygen deprivation and cortical infarction. CSD may therefore complement vasospasm as a mechanism of delayed ischemic lesions and neuro-worsening. Pathologic mechanisms and therapeutic implications of CSD will be explored in experimental and clinical research presented in joint sessions with the Co-Operative Study on Brain Injury Depolarizations (COSBID).
Cerebral vasospasm is the leading potentially treatable cause of death and disability after aneurysmal rupture. Almost 50% of patients who develop vasospasm will progress to cerebral infarction, and 15-20% will develop a disabling stroke or die, despite maximal therapy. With improved management of SAH patients, vasospasm has significantly decreased as a cause of death during the last decades however, rescue therapies for vasospasm are by no means completely effective and additional treatments are needed.
Advancements in neurological monitoring and neurocritical care management likely play a significant role in the reduction in death due to CV after SAH. Following aggressive, early neurosurgical or endovascular intervention to secure the aneurysm, appropriate neurocritical care management is essential to avoiding secondary injury that may adversely impact outcome. We will discuss novel approaches to neurological monitoring and neurocritical care in the management of events after subarachnoid hemorrhage.
We are pleased to welcome Dr. Tomio Sasaki, Professor and Chairman of the Department of Neurosurgery at the Kyushu University Graduate School of Medical Sciences as a keynote speaker for this aspect of the program.Throughout the program, we will be integrating presentations of basic science and translational research that contribute to clinical trial design and evidence-based management of SAH patients. We are very excited to welcome Dr. Constantino Iadecola, Chief of Division of Neurobiology at Cornell University, New York, as a keynote speaker. Each session will include a review of the current state of research in that area, and selected abstracts chosen for their innovation and potential impact. As well as platform sessions, there will be poster presentations. There will also be awards for best abstracts in three competition categories.
